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  • Ahsan N et al Comparative

    2020-08-18

    Ahsan, N., et al., 2018. Comparative effects of Bisphenol S and Bisphenol A on the development of female reproductive system in rats; a neonatal exposure study. Chemosphere 197, 336e343.
    Aiba, T., et al., 2018. Does the prenatal bisphenol A exposure alter DNA methylation levels in the mouse hippocampus?: an analysis using a high-sensitivity meth-ylome technique. Gene Environ. 40, 12.
    Alonso-Magdalena, P., et al., 2016. Bisphenol-A and metabolic diseases: epigenetic, developmental and transgenerational basis. Environ Epigenet 2 (3), dvw022.
    Bastos Sales, L., et al., 2013. Effects of endocrine disrupting chemicals on in vitro global DNA methylation and adipocyte differentiation. Toxicol. In Vitro 27 (6), 1634e1643.
    Bhan, A., et al., 2014. Histone methyltransferase EZH2 is transcriptionally induced by estradiol as well as estrogenic endocrine disruptors bisphenol-A and
    Chenais, B., 2015. Transposable elements in cancer and other human diseases. Curr.
    Cox, T.R., Erler, J.T., 2011. Remodeling and homeostasis of the extracellular matrix:
    implications for fibrotic diseases and cancer. Dis. Models Mech. 4 (2), 165.
    Deng, Q., et al., 2018. GPER/Hippo-YAP signal is involved in Bisphenol S induced migration of triple negative breast cancer (TNBC) cells. J. Hazard Mater. 355, 1e9.
    Doerks, T., et al., 2002. Systematic identification of novel protein domain families associated with nuclear functions. Genome Res. 12 (1), 47e56.
    Doherty, L.F., et al., 2010. In utero exposure to diethylstilbestrol (DES) or bisphenol-A (BPA) increases EZH2 expression in the mammary gland: an epigenetic mechanism linking endocrine disruptors to breast cancer. Hormones Canc. 1 (3), 146e155.
    Gingrich, J., et al., 2018. Gestational bisphenol S impairs placental endocrine func-tion and the fusogenic trophoblast signaling pathway. Arch. Toxicol. 92 (5), 1861e1876.
    Graff, J.R., et al., 2000. Methylation patterns of the E-cadherin 5' CpG island are unstable and reflect the dynamic, heterogeneous loss of E-cadherin expression during metastatic progression. J. Biol. Chem. 275 (4), 2727e2732.
    Hu, T., et al., 2017. Hypermethylated LTR retrotransposon exhibits enhancer activity.
    Huang, D.W., et al., 2007. The DAVID Gene Functional Classification Tool: a novel biological module-centric algorithm to functionally analyze large gene lists. Genome Biol. 8 (9). R183-R183.
    Ideta-Otsuka, M., et al., 2017. Epigenetic toxicity of environmental chemicals upon exposure during development - bisphenol A and valproic 96187-53-0 may have epigenetic effects. Food Chem. Toxicol. 109 (Pt 1), 812e816.
    Konkel, L., 2013. Thermal reaction: the spread of bisphenol S via paper products.
    Lander, E.S., et al., 2001. Initial sequencing and analysis of the human genome.
    Lu, P., et al., 2012. The extracellular matrix: a dynamic niche in cancer progression.
    Machtinger, R., et al., 2018. Urinary concentrations of phthalate metabolites, bisphenols and personal care product chemical biomarkers in pregnant women in Israel. Environ. Int. 116, 319e325.
    Martin, E.M., Fry, R.C., 2018. Environmental influences on the epigenome: exposure-associated DNA methylation in human populations. Annu. Rev. Public Health 39, 309e333.
    Mlynarcikova, A., et al., 2013. Bisphenol A alone or in combination with estradiol modulates cell cycle- and apoptosis-related proteins and genes in MCF7 cells. Endocr. Regul. 47 (4), 189e199.
    Wan, Y., et al., 2018a. Relationship between maternal exposure to bisphenol S and pregnancy duration. Environ. Pollut. 238, 717e724. Wan, Y., et al., 2018b. Spatial distribution of bisphenol S in surface water and human serum from Yangtze River watershed, China: implications for exposure through drinking water. Chemosphere 199, 595e602.
    Wang, W., et al., 2015. A comparative assessment of human exposure to tetra-bromobisphenol A and eight bisphenols including bisphenol A via indoor dust ingestion in twelve countries. Environ. Int. 83, 183e191.
    Wolstenholme, J.T., et al., 2013. Transgenerational effects of prenatal bisphenol A on social recognition. Horm. Behav. 64 (5), 833e839. Xin, F., et al., 2018. Mice exposed to bisphenol A exhibit depressive-like behavior with neurotransmitter and neuroactive steroid dysfunction. Horm. Behav. 102, 93e104.
    Contents lists available at ScienceDirect
    The Breast
    Original article
    Bisphosphonate use and incident cardiovascular events among older breast cancer survivors
    Nana Gegechkori a, d, *, Natalia Egorova b, Grace Mhango a, Juan P. Wisnivesky a, c, Jenny J. Lin a
    a Division of General Internal Medicine, Icahn School of Medicine at Mount Sinai, One Gustave L. Levy Place, Box 1087, New York, NY, 10029, USA
    b Department of Population Health Science and Policy, Icahn School of Medicine at Mount Sinai, One Gustave L. Levy Place, New York, NY, 10029, USA